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Agent-induced gastric lesion models , a cytotoxic agent producing stomach and duodenal lesions, where the damage was framed as a nitric oxide system disturbance Cyclophosphamide , producing stomach lesions in mice, with BPC-157 showing attenuation alongside omeprazole and bromocriptine while several other agents did not Haloperidol , relevant since non-steroidal anti-inflammatory drugs are among the most common causes of gastric mucosal injury NSAID-type damage The proposed mechanism combines increased mucosal perfusion with direct cytoprotection, consistent with the broader mechanistic picture
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